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Bibliographic Details
Format: Book
Published: dic. 2005
Online Access:http://www.scielo.cl/scielo.php?script=sci_arttext&pid=S0034-98872005001200011&lng=pt&nrm=iso&tlng=es
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fullrecord oai:netcat:be-dblil-85 BR1.1 LILACS LILACSEXPRESS MEDLINE SP as Internet <i>http://www.scielo.cl/scielo.php?script=sci_arttext&pid=S0034-98872005001200011&lng=pt&nrm=iso&tlng=es</i> Nuevos receptores nucleares heterodimÚricos: reguladores metabólicos con impacto en fisiopatologÝa y su proyección terapÚutica en dislipidemias y diabetes mellitus New heterodimeric nuclear receptors: key metabolic regulators with relevance in the pathophysiology and therapy of dyslipidemias and diabetes mellitus Rev. mÚd. Chile 133 12 0034-9887 ilus tab Es En Fondo Nacional de Desarrollo CientÝfico y Tecnológico de Chile 1030416 dic. 2005 20051200 57 The regulation of gene expression is crucial for the normal development and the homeostatic maintenance of body tissues. Thus, its malfunction may determine a variety of human disease conditions. A growing body of evidence has shown the overwhelming relevance of a new class of gene expression regulators: the heterodimeric nuclear receptors, a family of structurally related proteins involved in multiple biological functions. In response to activating ligands, these molecules bind to specific genomic regulatory regions where they can coordinately modify the transcriptional activity of several genes involved in the main metabolic pathways of lipids and carbohydrates in cells. These functional properties have stimulated the study of the relationships between heterodimeric nuclear receptors and various disease conditions, such as dyslipidemias and diabetes mellitus. Here we review the experimental, clinical and epidemiological evidences that support the relevance of these transcriptional regulators in the pathophysiology of the most prevalent and lethal diseases in Western countries. We also explore the potential therapeutic impact of new strategies based in the pharmacological modulation of the heterodimeric nuclear receptors.(AU) 2006-06-09 20060531 SCIELO JA FONTE 428533 <d>BIREME_LXP</d> <s>S0034-98872005001200011</s> CortÚs, VÝctor <s1>Pontificia Universidad Católica de Chile</s1> <s2>Facultad de Medicina</s2> <s3>Departamentos de GastroenterologÝa</s3> <c>Santiago</c> <p>Chile</p> Quezada, Nicolßs <s1> Pontificia Universidad Católica de Chile</s1> <c>Santiago</c> <p>Chile</p> Rigotti, Attilio <s1>Pontificia Universidad Católica de Chile</s1> <s2>Facultad de Medicina</s2> <s3>Departamentos de GastroenterologÝa</s3> <c>Santiago</c> <p>Chile</p> Maiz, Alberto <s1>Pontificia Universidad Católica de Chile</s1> <s2>Facultad de Medicina</s2> <s3>Departamento de Nutrición, Diabetes y Metabolismo</s3> <c>Santiago</c> <p>Chile</p> <f>1483</f> <l>1492</l> 85
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publishDate dic. 2005
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url http://www.scielo.cl/scielo.php?script=sci_arttext&pid=S0034-98872005001200011&lng=pt&nrm=iso&tlng=es